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Some of the AVH patients included in the current study had been reported with the case history, EEG, neuroimaging and the MEG findings. In these previous studies, the AVH patients had been confirmed to have some brain structural anomalies with the EEG, the neuroimaging. In our study, the same patients presented abnormal MEG findings of increased fMRI amplitude and decreased fMRI latency for the AVH compared with that of the controls. The result of this study is consistent with the previous studies. Therefore, MEG can be a complementary measure to fMRI.

In patients with clinical epilepsy, the AVH at the seizure onset in temporal lobe epilepsy can be localized by MEG and EEG.^[@B28]^ Some studies have reported that in AVH patients, the intensity of AVH is related to the focus size of the epileptic discharge.^[@B29],[@B30]^ Further studies are needed in order to determine if the brain areas are different between AVH patients and controls when the frequency of the AVH is controlled.

Some limitations to this study should be addressed. The first of these is the small size of the study. Second, the fMRI data have been generated with a different type of filter system, and the physiological noise will be different. Third, the epileptiform discharges in the EEG were not considered in this study. Finally, we cannot exclude the possibility that the AVH patients may be different from the other patients without AVH, since the AVH patients were recruited from the only psychiatric unit in the hospital.

Conclusions

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